In 1999 Jerry Brunetti was diagnosed with Non-Hodgkin’s Lymphoma and given 6 months to live. He did not submit to chemotherapy, but rather, developed his own unique dietary approach to enhance his immune system. In this informative video, Jerry shares his personal experiences and provides his recipe for healthy living. You will learn about the crucial importance of minerals, which foods to choose for your best health requirements and what to avoid. After viewing this video you’ll realize the remarkable value of food in building good foundations, and providing buffers, to keep your body healthy.
Topics of the second video include:
- The virtue of Cilantro or coriander in mopping up heavy metals in your system as it’s a very effective heavy metal chelator.
- The virtue of salicilic acid.
- Why eggs are so healthy.
- Coconut oil, an extremely healthy and healing oil.
- Why cholesterol isn’t bad of and by itself.
- Flax seed oil, a great source of omega 3 fatty acids.
- The trouble with grains and the benefits of fermented grain (sourdough bread)
- Good milk versus bad milk; hyper immune milk and raw milk versus pasteurized and homogenized milk
- Good things about butter.
- Good things about cholesterol.
- Good soy (=fermented soy), Bad soy (=non-fermented soy, such as soy milk).
- The virtue of selenium and iodine.
- Natural anti-cancerous compounds.
Check out the accompanying resources page for slides and food advice.
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Dodecanol, also known by its IUPAC name 1-dodecanol or dodecan-1-ol, and by its trivial name dodecyl alcohol and lauryl alcohol, is a fatty alcohol. Dodecanol is a colourless, water insoluble solid of melting point 24 °C and boiling point 259 °C. It has a floral odor. It can be obtained from palm kernel or coconut oil fatty acids and methyl esters by reduction.
In cosmetics, dodecanol is used as an emollient.
Traveler’s diarrhea (in American English) or traveller’s diarrhœa (in British English), abbreviated to TD, is the most common illness affecting travelers. Traveler’s diarrhea is defined as three or more unformed stools in 24 hours passed by a traveler, commonly accompanied by abdominal cramps, nausea, and bloating. It does not imply a specific organism, but enterotoxigenic Escherichia coli is the most common.
Wilderness diarrhea (WD), also called wilderness-acquired diarrhea (WAD) or backcountry diarrhea, is the name preferred by some backpackers, hikers, campers and other outdoor recreationalists for traveler’s diarrhea that appears in wilderness or “backcountry” situations while still in their home country. It is due to the same agents as all other traveler’s diarrhea, which are usually bacterial and viral in short expeditions and may be giardiasis in longer expeditions. and is largely due to the absence of treated water and poor hygiene. Some people reserve the name backpacker’s diarrhea as a synonym for giardiasis.
The primary source of infection is ingestion of fecally contaminated food or water.
The most important determinant of risk is the traveler’s destination. High-risk destinations are the developing countries of Latin America, Africa, the Middle East, and Asia. Some first world countries are also deemed risky. A worldwide rating of drinking water safety is kept at Safe Water for International Travelers website.
Among backpackers, additional risk factors for this class of infections include drinking untreated surface water and failure by the individual and his or her companions to maintain personal hygiene practices and clean cookware. Campsites often have very primitive sanitation facilities, if any, making them as potentially dangerous as any third-world country.
People at particular high-risk include young adults, immunosuppressed persons, persons with inflammatory-bowel disease or diabetes, and those taking H-2 blockers or antacids. Attack rates are similar for men and women.
Although traveler’s diarrhea usually resolves within three to five days (mean duration: 3.6 days), in about 20 percent of persons the illness is severe enough to cause bed confinement and in 10 percent of cases the illness lasts more than one week.
Salicylic acid (SA) is a phytohormone; and a phenol,is found everywhere in plants generating a significant impact on plant growth and development, photosynthesis, transpiration, ion uptake and transport and also induces specific changes in leaf anatomy and chloroplast structure. SA is recognized as an endogenous signal, mediating in plant defense, against pathogens It plays a role in the resistance of pathogens by inducing the production of ‘pathogenesis-related proteins’. It is involved in the systemic acquired resistance [SAR] in which a pathogenic attack on older leaves causes the development of resistance in younger leaves, though whether SA is the transmitted signal is debatable.
Medicinal and cosmetic uses
Also known as 2-hydroxybenzoic acid, one of several beta hydroxy acids (compare to AHA), salicylic acid is a key ingredient in many skin-care products for the treatment of acne, psoriasis, calluses, corns, keratosis pilaris, and warts. It works by causing the cells of the epidermis to shed more readily, preventing pores from clogging up, and allowing room for new cell growth. Because of its effect on skin cells, salicylic acid is used in several shampoos used to treat dandruff. Salicylic acid is also used as an active ingredient in gels that remove warts. Use of concentrated solutions of salicylic acid may cause hyperpigmentation on unpretreated skin for those with darker skin types (Fitzpatrick phototypes IV, V, VI), as well as with the lack of use of a broad spectrum sunblock.
Subsalicylate in combination with bismuth form the popular stomach relief aid known commonly as Pepto-Bismol. When combined, the two key ingredients help control diarrhoea, nausea, heartburn, and gas. It is also a very mild antibiotic.
[...]vegetarians have high blood levels of salicylic acid, the active ingredient in aspirin for treating inflammation. The authors suggest that this may help explain why people who have a diet rich in fruits and vegetables tend to have a lower incidence of heart disease and cancer.
Aspirin, which is acetylsalicylic acid, hydrolyzes to salicylic acid 20 minutes after entering the bloodstream. Salicylic acid, a compound that plays an important role in resisting pathogen infection in plants, is found in varying quantities in all fruits, vegetables, herbs, and spices.
Fruits with a high salicylate content are raisins, prunes, raspberries, apricots, blackberries, blueberries, boysenberries, cantaloupe, cherries, cranberries, currants, dates, guava, grapes, loganberries, oranges, pineapples, plums, and strawberries.
Vegetables with a high salicylate content are broccoli, chili peppers, cucumber, okra, spinach, squash, sweet potato, canned tomatoes, tomato paste and sauce, green peppers, radishes and zucchini.
The highest concentrations of salicylic acid are found in herbs and spices. These include aniseed, canella powder, cayenne, celery powder, cinnamon, curry, dill powder, fenugreek powder, garam masala, mustard powder, oregano, paprika, rosemary, sage, tarragon, turmeric, thyme, mint, black pepper, bay leaves, basil, caraway, ginger root, and nutmeg (2). It is also found in honey and Worcestershire sauce.
Non-steroidal anti-inflammatory drugs, usually abbreviated to NSAIDs or NAIDs , are drugs with analgesic, antipyretic and, in higher doses, anti-inflammatory effects – they reduce Pain, fever and inflammation. The term “non-steroidal” is used to distinguish these drugs from steroids, which (among a broad range of other effects) have a similar eicosanoid-depressing, anti-inflammatory action. As analgesics, NSAIDs are unusual in that they are non-narcotic. NSAIDs are sometimes also referred to as non-steroidal anti-inflammatory agents/analgesics (NSAIAs) or non-steroidal anti-inflammatory medicines (NSAIMs). The most prominent members of this group of drugs are aspirin, ibuprofen, and naproxen partly because they are available over-the-counter in many areas.
NSAIDs are usually indicated for the treatment of acute or chronic conditions where pain and inflammation are present. Research continues into their potential for prevention of colorectal cancer, and treatment of other conditions, such as cancer and cardiovascular disease.
NSAIDs are generally indicated for the symptomatic relief of the following conditions: (Rossi, 2006)
- Rheumatoid arthritis
- Inflammatory arthropathies (e.g. ankylosing spondylitis, psoriatic arthritis, Reiter’s syndrome)
- Acute gout
- Dysmenorrhoea (menstrual pain)
- Metastatic bone pain
- Headache and migraine
- Postoperative pain
- Mild-to-moderate pain due to inflammation and tissue injury
- Pyrexia (fever)
- Renal colic
- They are also given to neonate infants whose ductus arteriosus is not closed within 24 hours of birth
Aspirin, the only NSAID able to irreversibly inhibit COX-1, is also indicated for inhibition of platelet aggregation. This is useful in the management of arterial thrombosis and prevention of adverse cardiovascular events. Aspirin inhibits platelet aggregation by inhibiting the action of thromboxane -A.
In 2001 NSAIDs accounted for 70,000,000 prescriptions and 30 billion over-the-counter doses sold annually in the United States (Green, 2001).
Senescence encompasses all of the biological processes of a living organism‘s approaching an advanced age (i.e., the combination of processes of deterioration which follow the period of development of an organism). The word senescence is derived from the Latin word senex, meaning “old man” or “old age” or “advanced in age”.
Cellular senescence is the phenomenon where normal diploid differentiated cells lose the ability to divide, normally after about 50 cell divisions in vitro, some cells become senescent before because of DNA double strand breaks, toxins etc. This phenomenon is also known as “replicative senescence”, the “Hayflick phenomenon”, or the Hayflick limit in honour of Dr. Leonard Hayflick who was the first to publish this information in 1965. In response to DNA damage (including shortened telomeres) cells either age or self-destruct (apoptosis, programmed cell death) if the damage cannot be repaired. In this ‘cellular suicide’, the death of one, or more, cells may benefit the organism as a whole. For example, in plants the death of the water-conducting xylem cells (tracheids and vessel elements) allows the cells to function more efficiently and so deliver water to the upper parts of a plant.
Aging of the whole organism
Organismal senescence is the aging of whole organisms. The term aging has become so commonly equated with senescence that the terms will be used interchangeably in this article. Aging is generally characterized by the declining ability to respond to stress, increasing homeostatic imbalance and increased risk of aging-associated diseases. Because of this, death is the ultimate consequence of aging. Differences in maximum life span among species correspond to different “rates of aging”. For example, inherited differences in the rate of aging make a mouse elderly at 3 years and a human elderly at 90 years. These genetic differences affect a variety of physiological processes, including the efficiency of DNA repair, antioxidant enzymes, and rates of free radical production.
Some animals, such as some reptiles and fish, age slowly. Some even exhibit “negative senescence”, in which mortality falls with age, in disagreement with the Gompertz-Makeham “law”.
Whether replicative senescence (Hayflick limit) plays a causative role in organismal aging is at present an active area of investigation.